Tuesday, 16 October 2012

Jonsson Cancer Center researchers discover mechanism to revive hope in promising lymphoma treatment

Researchers at UCLA’s Jonsson Comprehensive Cancer Center have discovered the mechanism by which an experimental drug, GCS-100, removes a protein from lymphoma cells that prevents the cells from responding to chemotherapy. This discovery revives hope in GCS-100, a drug that had begun in clinical trials years before but had been delayed indefinitely. The researchers hope GCS-100 can be combined with chemotherapy to create an effective treatment for diffuse large B cell lymphoma (DLBCL), the most common and aggressive form of non-Hodgkin lymphoma, a cancer of the immune system. Read more here.

Study mentioned: Clark MC, et al. Galectin-3 binds to CD45 on diffuse large B cell lymphoma cells to regulate susceptibility to cell death. Blood. 2012 Oct 12. [Epub ahead of print] PMID: 23065155

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